Pathophysiology of Lung Cancer & COPD

Pathophysiology of Lung Cancer & COPD Photo

Inflammation is present in the lungs of people who smoke. This response to the inhaled toxins is increased in COPD, leading to tissue destruction, impairment of the defence mechanisms and disruption of the repair mechanisms. In general, the inflammatory and structural changes in the airways increase with disease severity and persist even after the smoking cessation. Apart from inflammation, two processes are involved in the pathogenesis of COPD—an imbalance between proteases and antiproteases and an imbalance between oxidants and antioxidants in the lungs.

These in turn result in various physiological diseases:

·         Tuberculosis

·         Pneumonia

·         Bronchiolitis

·         Ciliary dysfunction or primary ciliary dyskinesia (PCD)

·         Pulmonary hypertension

·         Hyperinflation

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